Home - Products - Others - Other Targets - N-terminally acetylated Endomorphin-1

N-terminally acetylated Endomorphin-1

CAS No.

N-terminally acetylated Endomorphin-1 ( Ac-L-Tyr-L-Pro-L-Trp-L-Phe-CONH2 )

Catalog No. M29950 CAS No.

N-terminally acetylated Endomorphin-1 is a modified Endomorphin-1.

Purity : >98% (HPLC)

COA Datasheet HNMR HPLC MSDS Handing Instructions
Size Price / USD Stock Quantity
100MG Get Quote Get Quote
200MG Get Quote Get Quote
500MG Get Quote Get Quote

Biological Information

  • Product Name
    N-terminally acetylated Endomorphin-1
  • Note
    Research use only, not for human use.
  • Brief Description
    N-terminally acetylated Endomorphin-1 is a modified Endomorphin-1.
  • Description
    N-terminally acetylated Endomorphin-1 is a modified Endomorphin-1.
  • Synonyms
    Ac-L-Tyr-L-Pro-L-Trp-L-Phe-CONH2
  • Pathway
    Others
  • Target
    Other Targets
  • Recptor
    ——
  • Research Area
    ——
  • Indication
    ——

Chemical Information

  • CAS Number
    --
  • Formula Weight
    652.7
  • Molecular Formula
    C36H40N6O6
  • Purity
    >98% (HPLC)
  • Solubility
    ——
  • SMILES
    ——
  • Chemical Name

Shipping & Storage Information

  • Storage
    (-20℃)
  • Shipping
    With Ice Pack
  • Stability
    ≥ 2 years

Reference

molnova catalog
related products
  • Grandifloroside

    Grandifloroside shows good antioxidative and potent anti-inflammatory activities, it also exhibits potent inhibitory activity against TNF-α and 5α-reductase, suggests that it might be developed as a source of potent anti-oxidative and anti-inflammatory agents and therapeutic agent for benign prostatic hypertrophy (BPH).

  • β-lonone

    β-lonone is an end-ring analog of β-carotenoids which widely distributed in fruit and vegetableswith?anti-proliferative anti-metastatic.

  • G-Protein antagonist...

    Substance P-related peptide that inhibits binding of G proteins to their receptors. Competitively and reversibly inhibits M2 muscarinic receptor activation of Gi or Go and inhibits Gs activation by β-adrenoceptors.